Progress in Pediatric Cardiology
Volume 24, Issue 2 , Pages 89-99 , January 2008

Oxygen Pressure Field Theory: A detailed description of vital gas exchange, at the capillary level for perfusionists

  • Gary Grist

      Affiliations

    • Corresponding Author InformationTel.: +1 816 234 3004; fax: +1 816 802 1245.

References 

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  2. Krogh A. The number and distribution of capillaries in muscles with calculation of the oxygen pressure head necessary for supplying the tissue. J Physiol Lond. 1918;52:409–415
  3. Kreuzer F. Oxygen supply to tissues: the Krogh model and its assumptions. Experientia. 1982;38(12):1415–1426
  4. Lubbers D. Fundamentals and significance of local oxygen pressure measurements and pO2 histograms in the evaluation of oxygen supply to organs and organisms.. In:  Ehrly AM editors. From determination of tissue oxygen pressure in patients. Oxford, New YorK: Pergamon Press; 1983;p. 1–13
  5. Lund N. Skeletal muscle surface oxygen pressure fields in normal human volunteers and in critically ill patients. In:  Ehrly AM editors. Determination of tissue oxygen pressure fields in patients. Oxford, New York: Pergamon Press; 1983;p. 53–59
  6. Popel AS. Theory of oxygen transport to tissue. Crit Rev Biomed Eng. 1989;17(3):257–321
  7. Schumacker PT, Samsel RW. Analysis of oxygen delivery and uptake relationships in the Krogh tissue model. J Appl Physiol. 1989;67(3):1234–1244
  8. Hugot P, Sicsic JC, Schaffuser A, et al. Base deficit in immediate postoperative period of coronary surgery with cardiopulmonary bypass and length of stay in intensive care unit. Intensive Care Med. 2003;29(2):257–261
  9. Murray DM, Olhsson V, Fraser JI. Defining acidosis in postoperative cardiac patients using Stewart's method of strong ion difference. Pediatr Crit Care Med. 2004;5(3):240–245
  10. Johnson BA, Weil MH. Redefining ischemia due to circulatory failure as dual defects of oxygen deficits and of carbon dioxide excesses. Crit Care Med. 1991;19(11):1432–1438
  11. McCullough JN, Zhang N, Reich DL, et al. Cerebral metabolic suppression during hypothermic circulatory arrest in humans. Ann Thorac Surg. 1999;67(6):1895–1899discussion 1919–21
  12. Merino C. Formulas for temperature PaO2 correction. Anesth Analg. 1999;89(4):1065
  13. Pearl JM, Thomas DW, Grist G, et al. Hyperoxia for management of acid–base status during deep hypothermia with circulatory arrest. Ann Thorac Surg. 2000;70(3):751–755
  14. Gaynor JW, Nicolson SC, Jarvik GP, et al. Increasing duration of deep hypothermic circulatory arrest is associated with an increased incidence of postoperative electroencephalographic seizures. J Thorac Cardiovasc Surg. 2005;130(5):1278–1286
  15. Wong PC, Barlow CF, Hickey PR, et al. Factors associated with choreoathetosis after cardiopulmonary bypass in children with congenital heart disease. Circulation. 1992;86(5 Suppl):II118–II126
  16. Boldt J, Kling D, von Bormann B, et al. Extravascular lung water and hemofiltration during complicated cardiac surgery. Thorac Cardiovasc Surg. 1987;35(3):161–165
  17. Steinhorn DM. Termination of extracorporeal membrane oxygenation for cardiac support. Artif Organs. 1999;23(11):1026–1030
  18. Grist G, Thomas D. Blood anion gaps and venoarterial carbon dioxide gradients as risk factors in long-term extracorporeal support. J Extra Corpor Technol. 1997;29(1):6–10

PII: S1058-9813(07)00154-3

doi: 10.1016/j.ppedcard.2007.10.004

Progress in Pediatric Cardiology
Volume 24, Issue 2 , Pages 89-99 , January 2008

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